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Reduction of Risk PotentialICP > 20 mmHg

Increased Intracranial PressureNCLEX cheat sheet

Increased ICP is elevated pressure inside the rigid skull from swelling, bleeding, or excess CSF.

27 key facts 5 practice questions Reviewed Oct 2026
The 1-minute sheetCinnaRN

Signs

  • ↓ LOC = earliest sign
  • Cushing's triad = late, ominous
  • ↑ BP, ↓ HR, irregular RR

More

  • Unequal/fixed pupils, HA
  • Projectile vomiting, posturing
  • Widened pulse pressure

Do

  • HOB 30°, head midline
  • Mannitol, hypertonic saline
  • Avoid suction, clustering care

Avoid

  • No hip flexion, neck flexion
  • Prevent Valsalva, coughing
Key takeaway

Earliest sign: decreasing level of consciousness / restlessness

Before you read

Test yourself first: 5 questions

Question 1 of 5

A nurse is positioning a client in the side-lying position to prevent pressure injuries. At what angle should the client be placed to avoid direct pressure on the trochanter?

What is increased intracranial pressure?

Because the skull cannot expand, rising pressure compresses brain tissue and reduces perfusion. Normal ICP is about 5–15 mmHg, and a sustained pressure above 20 mmHg needs treatment; cerebral perfusion pressure (CPP = MAP − ICP) should stay at 60 mmHg or more. A change in level of consciousness is the earliest and most sensitive sign.

Increased Intracranial Pressure illustrated

Increased intracranial pressure: swollen brain, compressed ventricles and herniation through the foramen magnum
Increased intracranial pressure: swollen brain, compressed ventricles and herniation through the foramen magnum. Illustration: CinnaRN.

Key points to remember

  • Earliest sign: decreasing level of consciousness / restlessness
  • Cushing's triad (late): hypertension with widening pulse pressure, bradycardia, irregular respirations
  • Headache, vomiting (often projectile), and unequal or sluggish pupils
  • In infants: bulging fontanel and high-pitched cry
  • Common causes: head injury, stroke, brain tumor, bleeding in the skull, meningitis and hydrocephalus.
  • A dilated or sluggish pupil on the side of the lesion suggests pressure on cranial nerve III.
  • Decorticate (flexion) and then decerebrate (extension) posturing signal worsening brain compression.
  • A high carbon dioxide level dilates brain vessels and raises ICP, so oxygenation and ventilation are kept normal.

Signs & symptoms

  • ↓ LOC = earliest sign
  • Cushing's triad = late, ominous
  • ↑ BP, ↓ HR, irregular RR

Nursing priorities

  1. Elevate head of bed 30° with the head midline to promote venous drainage
  2. Keep the environment calm; avoid clustering care and anything that raises ICP
  3. Prevent Valsalva — avoid straining, coughing, and hip flexion
  4. Give osmotic diuretics (mannitol) as ordered; monitor neuro status closely
  5. Do neuro checks — Glasgow Coma Scale, pupils and vital signs — at the ordered frequency and report any decline.
  6. Keep the temperature normal; fever raises the brain's oxygen demand.
  7. Give stool softeners and antiemetics as ordered to prevent straining and vomiting.

Labs & values

ICP5–15 mmHg (treat sustained > 20 mmHg)
CPP = MAP − ICP, goal60 mmHg or more
Serum osmolality and sodium checked during mannitol or hypertonic saline (mannitol is often held when osmolality exceeds320 mOsm/kg)
515mmHg Normal Increased Intracranial Pressure

Patient teaching

  • Report worsening headache, vomiting, or vision changes promptly
  • Family: keep the room quiet, limit visitors and stimulation, and let the patient rest between care.
  • Avoid bearing down, forceful coughing, and bending at the waist.

Red flags: report now

  • Cushing's triad, fixed/dilated pupils, or sudden LOC drop — neurologic emergency
  • Avoid suctioning longer than 10 seconds and avoid Trendelenburg position
  • A drop in GCS of 2 or more points, a newly dilated pupil, or posturing — notify the provider immediately.

References

Clinical references for this topic

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