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Physiological AdaptationEnlarged prostate, weak stream

Benign Prostatic HyperplasiaNCLEX cheat sheet

Benign prostatic hyperplasia is a non-cancerous enlargement of the prostate gland common in aging men.

24 key facts 3 practice questions Reviewed Oct 2026
The 1-minute sheetCinnaRN

Signs

  • weak stream, hesitancy, dribbling
  • frequency, nocturia, urgency
  • retention, incomplete emptying

Meds

  • -osin (alpha block) → ↓BP, dizzy
  • finasteride: shrinks, takes wks

Report

  • acute retention = emergency
  • no void → hydronephrosis, AKI

Post-Turp

  • CBI: keep urine pink, not red
  • bright red + clots → bleeding
  • no straining, no heavy lifting
Red flag to remember

Acute inability to void with a distended, painful bladder - urinary retention emergency; report and catheterize.

Before you read

Test yourself first: 3 questions

Question 1 of 3

A client is admitted with suspected postrenal acute kidney injury. Which condition is the most likely cause?

What is benign prostatic hyperplasia?

The enlarged gland compresses the urethra, obstructing urine flow from the bladder. It matters because chronic obstruction can lead to urinary retention, recurrent infections, bladder distention, and kidney damage. It is not cancer, but symptoms overlap and PSA may be checked.

Benign Prostatic Hyperplasia illustrated

BPH: an enlarged prostate compressing the urethra
BPH: an enlarged prostate compressing the urethra. Illustration: BruceBlaus via Wikimedia Commons, CC BY-SA 4.0.

Key points to remember

  • Obstructive symptoms include weak/hesitant stream, dribbling, incomplete emptying, and straining to void.
  • Irritative symptoms include urinary frequency, urgency, and nocturia.
  • Urinary retention with bladder distention can develop, causing overflow incontinence.
  • Stasis of urine increases the risk of UTIs and bladder stones.
  • Chronic backup of urine can cause hydronephrosis and renal impairment.
  • Digital rectal exam reveals a smooth, firm, enlarged prostate.

Signs & symptoms

  • weak stream, hesitancy, dribbling
  • frequency, nocturia, urgency
  • retention, incomplete emptying

Nursing priorities

  1. Assess for urinary retention - palpate/scan for bladder distention and monitor output.
  2. Catheterize for acute retention as ordered, releasing urine gradually if extreme distention.
  3. Administer alpha-blockers (relax muscle, e.g., tamsulosin) and 5-alpha-reductase inhibitors (shrink gland, e.g., finasteride) as prescribed.
  4. Encourage scheduled voiding and adequate fluids; monitor post-void residuals.
  5. Post-prostatectomy/TURP: maintain continuous bladder irrigation and monitor output color.

Labs & values

PSA: normal under4 ng/mL (may be mildly elevated; rules out cancer)
BUN/Creatinine: normal unless obstruction causes renal damage
Urinalysis: may show infection from urinary stasis

Patient teaching

  • Avoid bladder irritants (caffeine, alcohol) and decongestants/anticholinergics that worsen retention.
  • Finasteride takes months to work and can lower PSA; alpha-blockers can cause dizziness, so change positions slowly.
  • Void on a schedule, do not delay urinating, and report inability to urinate immediately.

Red flags: report now

  • Acute inability to void with a distended, painful bladder - urinary retention emergency; report and catheterize.
  • After TURP, bright red bloody outflow with clots or sudden decreased irrigation outflow - report immediately.
  • Signs of obstruction causing renal impairment (decreasing output, rising creatinine) - escalate.
  • Tamsulosin can cause orthostatic hypotension - take fall precautions.

References

Clinical references for this topic

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