👤 By the CinnaRN Clinical Content Team🕐 Updated 2026-08-23🏷️ Physiological Adaptation🔖 Free to read, print, and share
Also known as: low potassium · low K · potassium deficit
Hypokalemia — medical illustration. Illustration: Open Courses, TEI of Athens via Wikimedia Commons, CC BY-SA 3.0.
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Use this quick-reference guide to spot, treat, and prevent Hypokalemia on the NCLEX. Keep it handy during review and on exam day!
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📒 The 1-minute cheat sheet
⚠️ Causes
Loop/thiazide diuretics, ↑ losses
Vomiting, diarrhea, NG suction
Insulin, alkalosis shift K+ in
🩺 Signs
Muscle weakness, leg cramps
↓ bowel sounds, ileus, constipation
ECG: flat T, U wave, ST↓
✅ Do
Give K+ slow → never IV push
Max 10 mEq/hr, on pump, diluted
↑ K+ diet: banana, potato, OJ
🚩 Report
Dig toxicity risk if low K+
Check renal/UO before replacing
📚 Hypokalemia — full study notes
The cheat sheet above is your quick recall card. These notes go deeper — what it is, what to do first, what must be reported, and what to teach.
Hypokalemia is a serum potassium below 3.5 mEq/L. Potassium is the major intracellular cation and is essential for normal cardiac, neuromuscular, and smooth-muscle function, so a deficit shows up first as muscle weakness and cardiac irritability. Common causes are diuretics (loop/thiazide), vomiting, diarrhea, NG suction, and insulin or alkalosis shifting K⁺ into cells.
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Key points
Understand these first
Muscle weakness, leg cramps, and fatigue — severe loss causes flaccid paralysis
Decreased GI motility → hypoactive bowel sounds, constipation, paralytic ileus
ECG changes: flattened T waves, ST depression, and prominent U waves
Increases the risk of digoxin toxicity — low K⁺ potentiates digoxin
Often paired with metabolic alkalosis (vomiting, diuretics)
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Nursing priorities
What to do, in order
Replace potassium — oral if mild; IV for severe (NEVER IV push) at no more than 10 mEq/hr
Always dilute IV potassium and use a pump; check that urine output is adequate first
Put the client on a cardiac monitor and watch for dysrhythmias
Hold further potassium-wasting diuretics and notify the provider
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Red flags — report now
Escalate immediately
K⁺ < 2.5 mEq/L or any dysrhythmia — notify provider immediately
Never give potassium IV push or as a bolus — it can cause fatal cardiac arrest
If on a loop/thiazide diuretic, report muscle weakness or palpitations
❓ Hypokalemia: NCLEX FAQs
What are the priority nursing interventions for Hypokalemia?
Replace potassium — oral if mild; IV for severe (NEVER IV push) at no more than 10 mEq/hr. Always dilute IV potassium and use a pump; check that urine output is adequate first. Put the client on a cardiac monitor and watch for dysrhythmias. Hold further potassium-wasting diuretics and notify the provider.
What are the warning signs of Hypokalemia a nurse must report?
K⁺ < 2.5 mEq/L or any dysrhythmia — notify provider immediately. Never give potassium IV push or as a bolus — it can cause fatal cardiac arrest.
What do I need to know about Hypokalemia for the NCLEX?
Muscle weakness, leg cramps, and fatigue — severe loss causes flaccid paralysis. Decreased GI motility → hypoactive bowel sounds, constipation, paralytic ileus. ECG changes: flattened T waves, ST depression, and prominent U waves. Increases the risk of digoxin toxicity — low K⁺ potentiates digoxin.
What patient teaching is important for Hypokalemia?
Eat potassium-rich foods: bananas, oranges, potatoes, avocado, spinach. If on a loop/thiazide diuretic, report muscle weakness or palpitations.
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Quick Tip
Muscle weakness, leg cramps, and fatigue — severe loss causes flaccid paralysis.