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Physiological AdaptationPump failure → ↓ CO

Cardiogenic ShockNCLEX cheat sheet

Cardiogenic shock is a state in which the heart cannot pump enough blood to meet the body's needs, most often after a large myocardial infarction.

22 key facts 5 practice questions Reviewed Oct 2026
The 1-minute sheetCinnaRN

Signs

  • ↓ BP, ↑ HR, weak pulse
  • Cool clammy skin, mottled
  • Crackles, ↓ urine, ↓ LOC

Report

  • High mortality, MODS
  • Pulmonary edema

Do

  • Inotropes: dobutamine, dopamine
  • O₂, diuretics, IABP/LVAD
  • Treat MI cause (PCI)

Causes

  • MI #1, HF, dysrhythmias
Key takeaway

It results from pump failure, most commonly after a massive MI, but also from severe heart failure, dysrhythmias, or valve failure.

Before you read

Test yourself first: 5 questions

Question 1 of 5

A nurse is caring for a client with cardiogenic shock following a large myocardial infarction. Which assessment finding would the nurse expect?

What is cardiogenic shock?

The failing pump causes a drop in cardiac output, poor tissue perfusion, and blood backing up into the lungs. It carries a high mortality, so the goal is to improve cardiac output and reduce the workload on the damaged heart.

Cardiogenic Shock illustrated

Cardiogenic Shock — medical illustration
Cardiogenic Shock — medical illustration. Illustration: Kislitsina et al. / Wikimedia Commons via Wikimedia Commons, CC BY 4.0.

Key points to remember

  • It results from pump failure, most commonly after a massive MI, but also from severe heart failure, dysrhythmias, or valve failure.
  • Signs include hypotension, tachycardia, cool clammy skin, weak pulses, and decreased urine output.
  • Pulmonary congestion with crackles and dyspnea occurs as blood backs up into the lungs.
  • Altered mental status and confusion reflect poor cerebral perfusion.
  • Unlike hypovolemic shock, the problem is the pump itself, not low blood volume, so heart sounds and pressures reflect fluid overload.

Common causes

  • MI #1, HF, dysrhythmias

Signs & symptoms

  • ↓ BP, ↑ HR, weak pulse
  • Cool clammy skin, mottled
  • Crackles, ↓ urine, ↓ LOC

Nursing priorities

  1. Ensure airway, breathing, and oxygenation, providing supplemental oxygen and preparing for possible intubation.
  2. Administer inotropes such as dobutamine to improve contractility and vasopressors to support blood pressure as ordered.
  3. Administer diuretics and vasodilators cautiously to reduce preload and afterload as ordered.
  4. Provide continuous hemodynamic and cardiac monitoring and monitor urine output hourly.
  5. Prepare for advanced support such as intra-aortic balloon pump or revascularization.

Labs & values

Lactate: elevated above1 mmol/L indicates poor tissue perfusion
Troponin: elevated reflecting the underlying MI
ABG showing metabolic acidosis and hypoxemia; BNP elevated

Patient teaching

  • Report chest pain, shortness of breath, or worsening symptoms immediately, as early treatment of MI prevents progression.
  • After recovery, follow the prescribed cardiac medication, diet, and activity plan to protect heart function.

Red flags: report now

  • Worsening hypotension, falling urine output, and deteriorating mental status indicate progressing shock requiring immediate action.
  • New or worsening pulmonary edema with severe dyspnea and hypoxia is an emergency.
  • Life-threatening dysrhythmias require immediate intervention.

References

Clinical references for this topic

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