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Physiological AdaptationHematemesis + melena = bleed

Upper GI BleedNCLEX cheat sheet

Upper GI bleeding is hemorrhage from the esophagus, stomach, or duodenum (proximal to the ligament of Treitz).

25 key facts 5 practice questions Reviewed Oct 2026
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Causes

  • PUD #1, varices, gastritis
  • NSAIDs, ASA, alcohol, stress

Signs

  • Hematemesis (coffee-ground/bright)
  • Melena = black tarry stool
  • ↓ BP, ↑ HR, ↓ H&H = shock

Do

  • 2 large-bore IVs, fluids/blood
  • NPO, NG tube, monitor VS
  • PPIs, endoscopy to find source

Report

  • Hgb <7, ongoing bleeding
  • Hypovolemic shock signs
Key takeaway

Coffee-ground emesis indicates blood altered by gastric acid; bright red hematemesis indicates active, brisk bleeding.

Before you read

Test yourself first: 5 questions

Question 1 of 5

A home care nurse reviews the chart of a client receiving warfarin. The most recent INR is 5.8. What is the nurse's PRIORITY action?

What is upper gi bleed?

Common causes are peptic ulcers, esophageal varices, gastritis, and Mallory-Weiss tears. It is a potential emergency because rapid blood loss leads to hypovolemic shock. Hallmark signs are hematemesis (bright red or coffee-ground emesis) and melena (black, tarry stools).

Upper GI Bleed illustrated

Cirrhosis backs blood up the portal vein into esophageal varices, which can rupture and bleed
Cirrhosis backs blood up the portal vein into esophageal varices, which can rupture and bleed. Illustration: CinnaRN.

Key points to remember

  • Coffee-ground emesis indicates blood altered by gastric acid; bright red hematemesis indicates active, brisk bleeding.
  • Melena (black, tarry, foul-smelling stool) signals blood that has been digested in the upper GI tract.
  • Early shock signs include tachycardia, hypotension, cool clammy skin, restlessness, and decreased urine output.
  • A rising BUN with normal creatinine reflects digestion and absorption of blood protein in the gut.
  • Esophageal varices from portal hypertension can produce massive, rapidly fatal hemorrhage.
  • Hemoglobin and hematocrit may initially appear normal and drop only after fluid shifts or resuscitation.

Common causes

  • PUD #1, varices, gastritis
  • NSAIDs, ASA, alcohol, stress

Signs & symptoms

  • Hematemesis (coffee-ground/bright)
  • Melena = black tarry stool
  • ↓ BP, ↑ HR, ↓ H&H = shock

Nursing priorities

  1. Assess airway, breathing, and circulation; establish two large-bore IV lines for fluid and blood replacement.
  2. Administer isotonic IV fluids and prepare to transfuse packed red blood cells per orders.
  3. Keep the patient NPO and prepare for endoscopy (EGD) to locate and treat the bleed.
  4. Monitor vital signs, level of consciousness, and intake/output frequently for signs of ongoing loss.
  5. Administer ordered proton pump inhibitors; for varices give octreotide first-line (vasopressin is largely outdated due to ischemic complications).

Labs & values

Hemoglobin:12-18 g/dL (males 14-18, females 12-16)
Hematocrit:37-52%
BUN:10-20 mg/dL (elevated with normal creatinine in upper GI bleed)
1218g/dL Normal

Patient teaching

  • Avoid NSAIDs, aspirin, alcohol, and smoking, which irritate the GI lining and worsen bleeding.
  • Report black tarry stools, vomiting blood, dizziness, or weakness immediately.
  • Take prescribed acid-suppressing medications as directed and complete any H. pylori treatment.

Red flags: report now

  • Hypotension, tachycardia, and altered mental status indicating hypovolemic shock require immediate intervention.
  • Bright red hematemesis or large-volume bleeding signals active hemorrhage and need for rapid escalation.
  • Sudden drop in hemoglobin/hematocrit with worsening vitals must be reported and treated immediately.

References

Clinical references for this topic

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